Lantow M, Schuderer J, Hartwig C, Simko M. (2006):

The goal of this study was to investigate whether radiofrequency (RFR) exposure at 1800 MHz causes production of free radicals and/or expression of heat-shock proteins in monocytes. Two cell types from human leukaemia cell lines were used. They were exposed to RFR, PMA, LPS, or heat. PMA and LPS can increase free radical production. Different modifications of the GSM signal were used – GSM-non DTX (speaking only), GSM-DTX (hearing only), and GSM-Talk (34% speaking and 66% hearing). The exposure was at SARs of 0.5, 1.0, 1.5, and 2.0 W/kg, for 45 minutes.

Heat and PMA induced a significant increase in superoxide radical anions and in reactive oxygen species (ROS) compared to sham or incubator controls. No significant differences were seen in free radical production after RFR exposure, either alone or combined with PMA or LPS exposure. The GSM-DTX signal produced a significant difference in free radical production when the data were compared to sham controls, but not when they were compared with incubator controls.

No significant effects were seen in hsp70 expression after the different RFR exposures.

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