Noviembre 2006
No
effect on well-being or cognitive performance from base station-like
exposure
This
study attempted to replicate the results of Zwamborn’s study
(2003), also known as the TNO study (see “What’s
New”, October 2003). That study reported that a UTMS base
station-like exposure at an E-field strength of 1 V/m reduced well-being
in hypersensitive and non-sensitive subjects. Regel and colleagues
from Switzerland could not find any effects on well-being from exposure
to a UTMS base station-like exposure. The study included subjects
that were self-reported to be sensitive to electromagnetic fields.
Effects were observed in 1 of 6 tasks of cognitive function in the
sensitive subjects and in accuracy in another task in non-sensitive
subjects. Both of these findings were apparently due to chance,
since they were not statistically significant after adjustment for
multiple testing.
For
more, see “Research –
Clinical – Others – General health effects”.
Reference:
Regel SJ, Negovetic S, Roosli M, Berdinas V, et al. UMTS base station-like
exposure, well-being, and cognitive performance. Environ Health
Perspect 2006;114:1270-1275.
Interphone
study (1): No increased risk of parotid gland tumours
This
study was based in Denmark and Sweden. Information was collected
from 60 malignant cases, 112 benign cases and 681 controls. For
regular phone use, regardless of duration, the risk estimates for
malignant and benign tumours were 0.7 (95% CI 0.4, 1.2) and 0.9
(0.5, 1.5) respectively. Similar results were found for more than
10 years' cell phone use. These results are in agreement with previous
reports.
Reference:
Lonn S, Ahlbom A, Christensen HC, Johansen C, et al. Mobile phone
use and risk of parotid gland tumor. Am J Epidemiol: Published on-line
July 3, 2006.
Interphone study (2): No increased
risk of acoustic neuroma
The
authors interviewed 101 cases of acoustic
neuroma aged 30-69 and residing in the Tokyo area, and 339 age-,
sex-, and residency-matched controls. No significant risk of acoustic
neuroma was observed with cell phone use. The OR
was 0.73 (CI 0.43-1.23). No exposure-related increase in the risk
was observed, and there was no association with laterality of the
tumours.
Reference:
Takebayashi T, Akiba S, Kikuchi Y, Taki M, et al. Mobile phone use
and acoustic neuroma risk in Japan. Occup Environ Med: Published
online 15 August 2006.
For
more on these studies, see "Research
- Epidemiology".
RFR from a GSM phone modifies brain excitability
Italian
investigators used Transmission Magnetic Stimulation to measure
brain excitability in volunteers who were exposed to RFR from a
900 MHz phone for 45 minutes. The results showed modification of
brain excitability, compared with sham exposure.
For
more, see "Research-Clinical-cognitive
function".
Reference:
Ferreri F, Curcio G, Pasqualetti P, De Gennaro L, et al. Mobile
phone emissions and human brain excitability. Ann Neurol 2006;60:188-196.
No effect on cell cycle or cell death from
RFR
A
recent study examined the effect of RFR from an 1800 MHz signal
on the cell cycle and on cell death, and showed no differences from
sham exposure.
For
more, see “Research –
Toxicological– others – cell cycle and cell death”.
Reference:
Lantow M, Viergutz T, Weiss DG, Simko M. Comparative study of cell
cycle kinetics and induction of apoptosis after exposure of human
mono Mac 6 cells to radiofrequency radiation. Radiat Res 2006;166:539-543.
Effects of RFR at high SARs on heat shock
proteins
Wang
and colleagues exposed human cells to RFR at 2450 MHz frequency
and high SAR levels for 3 hours. They found that HSP70 and HSP27
levels did not increase more than corresponding heat controls. A
transient increase in HSP27 phosphorylation was seen at SAR levels
above 100 W/kg, in comparison to heat controls.
For
more, see “Research –
Toxicological– others – heat shock response”.
Reference:
Wang J, Koyama S, Komatsubara Y, Suzuki Y, et al. (2006): Effects
of a 2450 MHz high-frequency electromagnetic field with a wide range
of SARs on the induction of heat-shock proteins in A172 cells. Bioelectromagnetics
27:479-486.
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