In 1980 Robinette et al. published results of a survey of Korean War veterans who had been exposed to radar. A summary of this paper can be found in "Research - Epidemiology". Now Groves and colleagues have reported on a forty-year follow-up of mortality due to cancer and other causes in the same group of Navy personnel. The results were similar to those of Robinette et al. - radar exposure had little effect on mortality.
This paper is only in abstract form.
Groves FD, Page WF, Gridley G, Lisimaque L, et al. (2002): Cancer in
Korean War Navy technicians: Mortality survey after forty years. Annals
of Epidemiology 12:488-534.
In "Research-Toxicological Experiments - Others" we discuss heat shock proteins (Hsp), which protect cells from a variety of stresses. There is some evidence that exposure to radiofrequency radiation (RFR) causes cells to produce these proteins. Another study has been published that supports that view. Shallom and colleagues at the Catholic University of America in Washington, DC, found that exposure to 915 MHz radiation led to an increased production of Hsp 70 compared to controls. They also found that chick embryos exposed to microwave radiation prior to being subjected to stress from reduced oxygen had better survival than non-exposed controls. The authors suggest that this increased survival may be due to Hsp production. The SARs in the experiment were higher than are found in cellular phone use. There was also a temperature increase in the experimental samples, although the authors did not feel that this accounted for the findings. The authors conclude that their study provides support for the hypothesis that "athermal EM field exposures induce Hsp 70 expression".
For more on this, see the section on this site referred to above.
Reference: Shallom JM, Di Carlo AL, Ko D, Penafiel LM, et al. (2002): Microwave exposure induces Hsp 70 and confers protection against hypoxia in chick embryos. Journal of Cellular Biochemistry 86:490-496.