Huang T-Q, Lee J-S, Kim T-H, Pack J-K, et al. (2005)

This is a study to determine whether RF radiation promotes tumour development in mice treated with a single application of dimethy;benz(a)anthracene (DMBA) to their shaved skin. DMBA is known to initiate tumourigenesis. The mice were randomized into four groups of 20. One group was sham-exposed, one was exposed to RFR at 849 MHz, another at 1,763 MHz, and a fourth was treated with 120O0tetradecoylphorbol-13-acetate (TPA), a tumour-promoting agent. The RFR-exposed groups were exposed at a whole-body average SAR of 0.4 W/kg for 2 cycles of 45 minutes with a 15 minute interval, 5 days a week for 19 weeks. The TPA group were given TPA twice weekly.

Tumours developed only in the TPA-treated group. There were no tumours in the other group, and follow-up for 1 year confirmed the absence of tumours. C-fos and cyclin D1 expression was only present in the skin of TPA-treated groups. C-fos is a gene involved in tumour initiation, and cyclin D1 is over-expressed in various human tumours and in other carcinogen-induced animal models.

The experiment was repeated using sham and RFR-exposed groups and the absence of tumours confirmed. The authors conclude:
"Our data suggest that 849 and 1,763 MHz radiations, similar to those emitted from mobile phones, do not have any promoting effect on skin tumour development in DMBA-initiated mice".

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